Berge, Anne Kristin McLaren

Anne Kristin McLaren Berge

Job description

Stipendiat i tumorbiologisk forskningsgruppe med tilknyttning til autofagiforskninsgruppen.

Scientific articles and book chapters
Other

Juncal Garcia Garcia, Anne Kristin McLaren Berge, Katrine Stange Overå, Kenneth Bowitz Larsen, Zambarlal Babanrao Bhujabal, Andreas Brech et al.:

TRIM27 is an autophagy substrate facilitating mitochondria clustering and mitophagy via phosphorylated TBK1

The FEBS Journal 2022 ARKIV / DOI

Synnøve Norvoll Magnussen, Kjersti Sellæg, Susannah Beatrice von Hofsten, Beate Hegge, Tonje Nygård Lillevik, Karoline Sollid Evensen et al.:

Hva gjør en kreftforsker?

2024

Anne Kristin Mclaren Berge, Kjersti Sellæg, Stine Figenschau, Saikat Das Sajib, Hallvard Lauritz Olsvik, Maria Perander et al.:

TRIM32 in Head and Neck Cancer

2023

Anne Kristin McLaren Berge, Juncal Garcia Garcia, Eva Sjøttem, Hallvard Lauritz Olsvik :

The ubiquitin E3 ligase TRIM27 emerges as a new player in mitophagy

Autophagy Reports 2023 ARKIV / DOI

Kjersti Sellæg, Susannah Beatrice von Hofsten, Anne Kristin McLaren Berge, Lennart Maximilian van Ligtenberg, Marcus Moe Mauseth, Athanasios Kournoutis et al.:

Autofagi: cellenes spareblussmodus

2023

Anne McLaren Berge :

Avfallshåndtering: fra store byer til små celler

UiT Forskerhjørnet 2022

Anne Kristin McLaren Berge :

Hvordan kan kreftcellene gjøres sårbare?

Forskersonen.no 2022

Research interests

I am working on a project that is focused on TRIM proteins in relation to autophagy and cancer. Autophagy is the renovation system of the cell and is essential for keeping our cells healthy. In cancer, autophagy is a double-edged sword as it can either prevent or promote the disease. Several TRIM proteins are already known to be involved in autophagy, but it remains to fully understand the participation of these proteins in the cells renovation system. Through bioinformatical methods in combination with wet-lab experiments, our goal is to know more about how TRIM proteins might be involved in cancer, either through autophagy or through a non-autophagic pathway.